Pulmonary Function Test Medical Transcription Sample Reports

Medical Transcription Pulmonary Function Test Sample Report #1

DATE OF STUDY: MM/DD/YYYY

REFERRING PHYSICIAN: John Doe, MD

INDICATION: Dyspnea.

INTERPRETATION OF STUDY: The patient’s spirometry showed small airway dysfunction. There was mild coving on the flow volume loops. Midexpiratory flows were 62% of predicted. Following the administration of an inhaled bronchodilator, there was no significant change in the patient’s airway mechanics. The patient’s FEV1 to FVC ratio was preserved at 78. Lung volume showed mild air trapping with residual volume of 156% of predicted by the nitrogen wash-out method. Maximum voluntary ventilation was normal, but diffusion capacity for carbon monoxide was substantially reduced at 54% of predicted.

IMPRESSION: Small airway dysfunction with mild air trapping and substantial reduction in the diffusion capacity. Clinical correlation was recommended.

Medical Transcription Pulmonary Function Test Sample Report #2

DATE OF STUDY: MM/DD/YYYY

REFERRING PHYSICIAN: John Doe, MD

HISTORY: Dyspnea.

INTERPRETATION OF STUDY: The patient’s spirometry is normal with no obstruction or small airway dysfunction. The FEV1 to FVC ratio is preserved at 80. Maximum voluntary ventilation is normal at 91% of predicted. The patient has a borderline restrictive ventilatory defect as measured by the nitrogen wash-out method with a total lung capacity of 78% of predicted. Diffusion capacity of carbon monoxide was mildly reduced at 60% of predicted. This interpretation of the patient’s diffusion capacity does not take into account the patient’s hemoglobin level (unavailable at the time of testing).

IMPRESSION: Borderline restrictive ventilatory defect with mild reduction of the diffusion capacity. Clinical correlation is recommended.

Medical Transcription Pulmonary Function Test Sample Report #3

DATE OF STUDY: MM/DD/YYYY

REFERRING PHYSICIAN: John Doe, MD

HISTORY: Dyspnea.

INTERPRETATION OF STUDY: The patient had a normal FEV1 of 1.67 liters or 86% of predicted. The forced vital capacity was also normal at 2.30 L or 92% of predicted. The FEV1 to FVC ratio was at the lower limits of normal at 72. Mid expiratory flows were reduced at 63% of predicted. The maximum voluntary ventilation was reduced to 45% of predicted. Following the administration of an inhaled bronchodilator, there was no significant change in the patient’s airway mechanics. The diffusion capacity for carbon monoxide was normal.

IMPRESSION: The flow volume loop and the isolated reduction in the expiratory flows at low lung volumes are consistent with small airway disease. Formal lung volume measurements are not available but a normal spirometric vital capacity argues against significant restriction. Following the inhalation of a bronchodilator, there was no significant change in airway obstruction. This does not necessarily mean that chronic bronchodilator therapy may not be useful. The reduction in the maximum voluntary ventilation is disproportionate to the patient’s FEV1 and reflects either the patient’s difficulty with the maneuver or neuromuscular dysfunction. The diffusion capacity for carbon monoxide is normal. Clinical correlation is recommended.